V08N03 | 2018 | Original Article | Pages 41 to 46
Luisa de Mello Florentino Guedes, Orlando Aguirre Guedes, Marcus Vinícius Corrêa da Costa, Artur Aburad de Carvalhosa, Cyntia Rodrigues de Araújo Estrela, Carlos Estrela
Periapical Periodontitis, Diagnosis, Epidemiology, Periapical Granuloma,
Introduction: The necrosis of the pulp tissue followed by the colonization and infection of the root canal space are determinant events to the installation, development and perpetuation of apical periodontitis. Objective: To evaluate the prevalence of different types of periapical inflammatory lesions of endodontic origin. Methods: The sample was composed of 805 histopathological reports issued and archived by the Oral Pathology Department of the Public Laboratory of Mato Grosso, Brazil between 2008 and 2014. The following information was collected from diagnosis requisition sheets and histopathological reports: gender, age, tooth group, anatomical localization and histopathological diagnosis (periapical granuloma, periapical abscess and radicular cyst). The statistical treatment analyzed data through frequency distribution and chi-square tests. The level of significance was set at 5% for all analyses. Results: A higher occurrence of periapical lesions was observed in males (n=405; 50.31%) and participants between 21 and 30 years old. The most affected teeth were the lower molars (n=293; 36.40%), followed by the upper molars (n=213; 26.46%). The periapical granuloma (n=458; 56.89%) was the most common periapical lesion. Statistically significant associations were observed between the type of periapical lesion and anatomical localization (p<0.05). Conclusions: There was a high prevalence of periapical lesions in males and participants between 21 and 30 years old. The periapical granuloma was the most commonly diagnosed periapical lesion, and the mandibular posterior teeth were the most affected teeth.
Guedes LMF, Guedes OA, Costa MVC, Carvalhosa AA, Estrela CRA, Estrela C. Prevalence of granulomas, abscesses and periapical cysts in inflammations of endodontic origin. Dental Press Endod. 2018 Sept-Dec;8(3):41-6. DOI: //doi.org/10.14436/2358-2545.8.3.041-046.oar